When I was eighteen, I had the Mirena IUD placed, which I finally removed a few months ago after reading your book and wanting to restore my hormone (as well as overall) health. I have also struggled with absent or extremely long cycles, which my doctors have told me is a result of PCOS. I am a 22 year-old student who has struggled with a restrictive eating disorder for over ten years.
Should i increase cals to 2500 and treat as HA frist then deal with high androgens? I have just started clare goodwins PCOS protocol and via questionnaire came out as ‘suspected type 1 (insulin resistance)’ but suspected, ! Serum androstenedione 7 nmol/L <6.00nmol/LĢ Plasma fasting glucose level – (HMA) – Normal – No Serum sex hormne binding glob 89 nmol/L 32.00 – 128.00nmol/L The typical therapeutic target for Children and AdolescentsĢ ! ANDROGENS – (ZH) – Suggestive of PCOS *Increased risk of significant hypoglycaemia with tight controlĬhildren and Adolescents (<18 years of age) Good Glycaemic Control HbA1c =59 mmol/mol Tight Glycaemic Control* HbA1c <48* mmol/mol Reference range applicable to morning samples collectedĢ HbA1c levl – IFCC standardised – (HMA) – Normal – And have tracke food macros iver past 5 years pretty consistent.Īlthough i appear to have a high LH to FSH ratio could i still have HA? (Attachdd are my bloods in april 2020 and repeat in august 2020)Ģ Serum cortisol – (HMA) – Normal – No Action 384 nmol/L 133.00 – 537.00nmol/L I am very active and eating about 1800 cals a day. No other symptoms.Ĭonfirmed on USS and bloods (high androgens) that i have PCOS. I have a question i would love you to answer.Ĭame off pill after 10 years nov 2020 and no period. What matters is how high or low it is compared to FSH.
LH increases with age so there is no “perfect” value for LH. When measured on day 2 of the cycle (or random day if there is no cycle), the LH to FSH ratio is high in PCOS and low in hypothalamic amenorrhea. The simplest way to distinguish between PCOS and hypothalamic amenorrhea is to look at the ratio between luteinizing hormone (LH) and follicle-stimulating hormone (FSH). And that’s a problem because the treatment for PCOS is to eat less while the treatment for hypothalamic amenorrhea is to eat more! In fact, relying on an ultrasound can result in hypothalamic amenorrhea being misdiagnosed as lean PCOS. PCOSĭo you notice that polycystic ovaries can occur with both PCOS and hypothalamic amenorrhea? That’s why PCOS cannot be diagnosed by ultrasound. Here’s a summary of the similarities and differences. The two conditions are similar enough that your doctor might mistakenly say you have PCOS when you actually have hypothalamic amenorrhea.
It can also present with mild acne, facial hair, and polycystic ovaries. Hypothalamic amenorrhea (HA) is the loss of periods due to undereating. PCOS is the condition of androgen excess when all other causes of androgen excess have been ruled out. What is the difference between polycystic ovary syndrome (PCOS) and hypothalamic amenorrhea?
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